已徵得 - 潤稿
By Agatha
at 2020-07-09T15:40
at 2020-07-09T15:40
Table of Contents
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[必]工 作 量: 12096 字(word)
[必]工作報酬: 整份英潤英 4500元
[必]涉及語言: 英文
[必]所屬領域: 生物 醫學
[必]文件類型: 提案
[必]截 稿 日: 20200715
[必]應徵期限: 徵到為止
[必]聯絡方式: 站內信
[必]付費方式: 承接工作後提供勞務報酬單,完稿後一周內由公司支付
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[選]工作要求: 文法糾正、語意正確即可。
[選]參考段落:
[選]試 譯 文: 請任選a或b段落潤稿(站內信)
a.
It was previously shown, that exosomes secreted by cancer cells will
sufficiently promote metastasis by various ways. For example exosomes derived
from breast cancer and prostate cancer cells induce neoplasia through
transfer of their miRNA cargo [14,15]. The plasticity of cancer cells may
also be attributed in part to exosomes, with exosomal miR-200 from metastatic
breast cancer cells enhancing the epithelial to mesenchymal transition (EMT)
and metastasis of otherwise weakly metastatic breast cancer cells [16]. EVs
participate in intercellular mitochondrial transfer. Horizontal transfer of
mitochondria occurs through the transfer of either mitochondria-derived
vesicles or intact mitochondria. To date, the mechanisms of intercellular
transfer of free mtDNA across both mitochondrial inner and outer membranes
and the plasma membrane remain elusive.
b.
Mitochondrial content, especially damaged or oxidized components resulting
from IR exposure, can act as damage-associated molecular patterns (DAMPs)
that activate an inflammatory response when present in the cytosol or
released from cells [43]. The mitochondrial antiviral signaling protein
(MAVS), located in the mitochondrial outer membrane, an innate immune
signaling molecule, is involved in radiation response via its oligomerization
mediated by radiation-induced ROS. MAVS is involved in IFN-β and
IFN-stimulated gene expression in the response to IR. MAVS complexes
promoting the nuclear translocation of the NF-κB and IRF3/7 transcription
factors, respectively, and thus elicit the innate antiviral response.
[選]其他事項:無
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* 如已讀過,請填 YES
* 如未讀過,請讀過再重新發文。未填視為未讀,一律刪文處理並警告。
──────────────────────────────────────
[必]工 作 量: 12096 字(word)
[必]工作報酬: 整份英潤英 4500元
[必]涉及語言: 英文
[必]所屬領域: 生物 醫學
[必]文件類型: 提案
[必]截 稿 日: 20200715
[必]應徵期限: 徵到為止
[必]聯絡方式: 站內信
[必]付費方式: 承接工作後提供勞務報酬單,完稿後一周內由公司支付
──────────────────────────────────────
[選]工作要求: 文法糾正、語意正確即可。
[選]參考段落:
[選]試 譯 文: 請任選a或b段落潤稿(站內信)
a.
It was previously shown, that exosomes secreted by cancer cells will
sufficiently promote metastasis by various ways. For example exosomes derived
from breast cancer and prostate cancer cells induce neoplasia through
transfer of their miRNA cargo [14,15]. The plasticity of cancer cells may
also be attributed in part to exosomes, with exosomal miR-200 from metastatic
breast cancer cells enhancing the epithelial to mesenchymal transition (EMT)
and metastasis of otherwise weakly metastatic breast cancer cells [16]. EVs
participate in intercellular mitochondrial transfer. Horizontal transfer of
mitochondria occurs through the transfer of either mitochondria-derived
vesicles or intact mitochondria. To date, the mechanisms of intercellular
transfer of free mtDNA across both mitochondrial inner and outer membranes
and the plasma membrane remain elusive.
b.
Mitochondrial content, especially damaged or oxidized components resulting
from IR exposure, can act as damage-associated molecular patterns (DAMPs)
that activate an inflammatory response when present in the cytosol or
released from cells [43]. The mitochondrial antiviral signaling protein
(MAVS), located in the mitochondrial outer membrane, an innate immune
signaling molecule, is involved in radiation response via its oligomerization
mediated by radiation-induced ROS. MAVS is involved in IFN-β and
IFN-stimulated gene expression in the response to IR. MAVS complexes
promoting the nuclear translocation of the NF-κB and IRF3/7 transcription
factors, respectively, and thus elicit the innate antiviral response.
[選]其他事項:無
──────────────────────────────────────
--
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By Bennie
at 2020-07-14T12:43
at 2020-07-14T12:43
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at 2020-07-18T15:36
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